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Myths vs Truths

drb_iac

Active member
There are few drugs in the world that are subjected to as much misinformation as anabolic steroids. When athletes were first using anabolic steroids in the 1930s > ’50s to break world records, the scientific and medical community simultaneously published studies that told us that they didn’t work. During that time the only legitimate information about these drugs was coming from the front lines; the weightlifters (and later bodybuilders) who had tried them and knew what they did. For decades there were scientists claiming anabolic steroids did not do anything to build muscle or strength and, to this day, the Physician’s Desk Reference says that they do not work to improve athletic performance.
Ultimately, medical science started playing catch up, but the damage was done and we steroid-users mostly turned a blind-eye to anything falling from the Ivory Towers of academia. And then came the Internet – where a hodge-podge of science and experience has been brought together to create some of the biggest steroid-myths we’ve ever seen. Unfortunately, since they come from other steroid users and not doctors, we’ve come to accept many of them as fact. And because the internet is the primary source of information on anabolic steroids at this point, the person who screams the loudest is the one who is most often repeated – right or wrong.

Myth: Steroids down-regulate your androgen receptors (this is why your first cycle is always your best).

Truth: The logic for this myth actually makes a lot of sense – receptor down-regulation is pretty obvious when you drink a cup of coffee every day for a month, then find you need to keep increasing the size to get the same “kick”. We see (and feel) this receptor downgrade with caffeine, clenbuterol, ephedrine and a ton of other stuff, so it’s logical to think that we’re seeing the same thing with steroids. Sadly, the science tells us otherwise. Steroids actually do the opposite – they up-regulate your androgen receptors. It’s wrong to think about androgen receptors as permanent receptacles for the androgen ligand (sort of like a fixed electrical outlet in your house). In reality, your androgen receptors are constantly being turned over. When unattached to an androgen they have a half life of approximately three hours and are ultimately replaced with new ones. However, in the presence of an androgen (i.e. when they’re attached), they become more sensitive, their half life is doubled and the amount of new receptors being formed also increases substantially. It’s also important to remember that AR-mediated effects are not the whole story when it comes to anabolic steroid activity in the body. There are still a host of other effects that have little to nothing at all to do with AR, known as non-AR dependent effects, which include central nervous system stimulation and a host of other anabolic and

potentially anabolic activities. But that still leaves us with the question of why our gains seem to slow down after a few cycles, and why we need to keep upping the dose. In truth, the answer probably has more to do with the body attempting to return to homeostasis through other mechanisms than it has with the androgen receptor per se. Still, if you’re worried about your androgen receptors you can try...no promises..some L-Carnitine L-Tartrate, a nutritional supplement that has been shown to increase androgen receptors (it was included in beastdrol, from NTBM for this exact reason).


Myth: Winstrol is the same whether you drink it or inject it.

Truth: When you pass a steroid through your liver it’s subject to a different metabolism to that of steroids injected directly into the muscle. In the case of Winstrol (Stanozolol), this includes greater interaction with SHBG, which lowers the amount of that carrier protein (allowing you to free up more androgen in the body). However, overall you get greater protein synthesis with the injectable route of administration, but both methods have their advantages. https://thinksteroids.com/articles/winstrol-oral-vs-injectable/ You can read more here, but injecting winstrol is called a first liver bypass. Seems science says injection is better. I have not found this to be so.


Myth: If you inject double the amount of testosterone you have twice as much in your bloodstream.

Truth: Again, this one seems to make a lot of sense. If you inject 600 milligrams of testosterone you should have twice as much in your blood as you would injecting 300 milligrams. But this isn’t how it works. The dose you’re taking isn’t equivalent to the blood plasma levels you’ll achieve – nor will doubling the dose necessarily double your blood plasma levels of testosterone. When scientists compared a 300 milligram shot of testosterone to a 600 milligram shot, they found that the 300mg weekly shot will get a normal male to approximately 1,345 ng/dl, while a 600mg weekly shot will get a normal male to 2,370 ng/dl (less than double the amount achieved with half the dose).


Myth: Testosterone is testosterone.

Truth: Although this seems like something that can’t be a myth, it is. Methyltestosterone (oral testosterone) converts to a much more potent version of oestrogen (methylestrogen) in the liver, while injectable testosterone does not. And, even the injectable testosterones have their differences; short esters convert to less oestrogen than longer esters. And, just to make things even more complicated, the patches and gels are convert to dihydrotestosterone to a much greater degree than either the injectable or oral testosterones.
Both oestrogen( I like to use the O...it is old school Oxford U!) and dihydrotestosterone have profound physiological effects on the body and, because every type of testosterone doesn’t convert to them equally, we find varying effects depending on the one we’re using. Oestrogen promotes greater adipose (fat) gain, but also greater muscle gain, while DHT gives a harder, more quality look to the physique. Still, although we see this “testosterone is testosterone” myth repeated (online
mostly) we also see top-flight bodybuilders taking advantage of the differences in various testosterones – long esters for bulking, short esters for cutting. So it’s not only the route of administration (oral, transdermal or injectable) that matters, but also the ester of the injectable versions. Testosterone is, strangely, not just testosterone.


Myth: Steroids don’t work unless you train and eat properly.

Truth: We’d love this to be true, wouldn’t we? But the truth is that numerous studies conducted on burn victims, the elderly and other special populations, tell us that anabolic steroids will work to build muscle even in the most catabolic conditions, and in the absence of training. We see the same thing with people who have terrible training programmes but continue to make gains because they’re juicing. For optimal results, steroids are added to a proper programme and diet, but they’ll help even if you’re doing everything wrong. Hey, I didn’t make the rules, I’m just telling you what the science says! Burn victims have sat in bed eating nasty ass hospital food and packed on some muscle. Sorry again ok?

Myth: The injection site doesn’t matter.

Truth: A study conducted by Minto et. al. in 1997 examined the differences between injecting in the deltoid (shoulder) versus the gluteus (butt). While we’d logically assume that if we’re injecting 100mgs of Nandrolone into the body it shouldn’t matter where the injection site is, but in reality nothing could be further from the truth. Injecting in the gluteus results in significantly higher blood plasma levels of the anabolic steroid in question. Put more simply, the men who received gluteal injections in the Minto study ended up with more Nandrolone in their bloodstream! In practical terms, this means if we’re going to inject one ml of anabolic steroid per week, we’d probably be getting the best results by alternating butt cheeks (or quads). For the guys doing a few ml per day, they’re going to have to stick themselves anywhere they’ve still got a piece of unscarred skin showing! Notice I said DECA...no ..sorry I said nandrolone but I meant deca. Well we always talk about those deep IM injections to leave the depot, to be slowly released. For some reason these long chain esters do better in bigger muscles. So lets do this. Long chains in the ass. TestE and cyp..where ever you want. Sus has some long chains and short chains...so if you really want to feel the shorts go delts...want to feel the decoanate go glutes




Myth: As long as the milligram amount is the same, the concentration doesn’t matter.

Truth: It seems like a reasonable assumption to say that a 100mg shot of Nandrolone is going to hit you the same, regardless of whether it’s 4ml of 25mg or 1ml of 100mg, right? Wrong. Minto (the guy from the myth above) took a look at Nandrolone volume per injection and compared it to final blood plasma levels, and found that the more highly concentrated shot (100mg in 1ml of oil versus 4ml) actually produced a higher concentration of steroid in the bloodstream. Why because the the less oil per mg means quicker crystal deposit, but this could also mean more pip. So for deca stay at 200mg/ml gear.


Myth: Equipoise (Bolednone) works similarly to Deca-Durabolin (Nandrolone).

Truth: These two anabolic steroids have been considered interchangeable in cycles since the first series of Underground Steroid Handbook updates were published (they were a newsletter, written by the late Dan Duchaine). When Dan first introduced Equipoise to the bodybuilding world, he stated that a quick look at it’s structure revealed that it probably worked something like Deca. Well, nothing could be further from the truth.
Deca is a progestin, an anabolic steroid derived from 19-nor testosterone, while Equipoise is simply testosterone with an additional double bond. If we look at their characteristics (conversion to oestrogenic metabolites, conversion to 5a-reduced metabolites), we find very few similarities between the two drugs. And while Equipoise has a reputation for increasing appetite, Deca lays claim to healing injuries and relieving joint pain. Deca is also likely to cause a bit more water retention at high mgs than Equipoise, although, when we talk in those kinds of terms we really start getting into a very subjective realm. The latest deca info is loking into the fact that deca causes increases of synovial fluid.
In reality deca and eq are pretty dissimilar, but because of a single error, made a couple of decades ago, we still see them being used interchangeably in cycles.

Ok my boys...you have a lot of reading to do, and I am heading back to costa rica. Hopefull Jake can answer questions for newbies! Take care all!
DrB
 
Very useful and relevant information that we want and are interested in. Just wondering drb your thoughts on any research data on LVH (Left Ventricle Hypertrophy) caused from long term use of AAS? I'm wondering if there may be a differentiating from long term therapeutical dosing versus long term supraphysiological dosing. I'm hoping that there may be something out there that shows low dosing, even for prolonged periods do not have impact on the shaping of the left ventricle, but of course that's me wishing for that data so that I can tell myself "We're good to go with low dosing". Just curios if you've seen more conclusive data on this.
 
There are few drugs in the world that are subjected to as much misinformation as anabolic steroids. When athletes were first using anabolic steroids in the 1930s > ’50s to break world records, the scientific and medical community simultaneously published studies that told us that they didn’t work. During that time the only legitimate information about these drugs was coming from the front lines; the weightlifters (and later bodybuilders) who had tried them and knew what they did. For decades there were scientists claiming anabolic steroids did not do anything to build muscle or strength and, to this day, the Physician’s Desk Reference says that they do not work to improve athletic performance.
Ultimately, medical science started playing catch up, but the damage was done and we steroid-users mostly turned a blind-eye to anything falling from the Ivory Towers of academia. And then came the Internet – where a hodge-podge of science and experience has been brought together to create some of the biggest steroid-myths we’ve ever seen. Unfortunately, since they come from other steroid users and not doctors, we’ve come to accept many of them as fact. And because the internet is the primary source of information on anabolic steroids at this point, the person who screams the loudest is the one who is most often repeated – right or wrong.

Myth: Steroids down-regulate your androgen receptors (this is why your first cycle is always your best).

Truth: The logic for this myth actually makes a lot of sense – receptor down-regulation is pretty obvious when you drink a cup of coffee every day for a month, then find you need to keep increasing the size to get the same “kick”. We see (and feel) this receptor downgrade with caffeine, clenbuterol, ephedrine and a ton of other stuff, so it’s logical to think that we’re seeing the same thing with steroids. Sadly, the science tells us otherwise. Steroids actually do the opposite – they up-regulate your androgen receptors. It’s wrong to think about androgen receptors as permanent receptacles for the androgen ligand (sort of like a fixed electrical outlet in your house). In reality, your androgen receptors are constantly being turned over. When unattached to an androgen they have a half life of approximately three hours and are ultimately replaced with new ones. However, in the presence of an androgen (i.e. when they’re attached), they become more sensitive, their half life is doubled and the amount of new receptors being formed also increases substantially. It’s also important to remember that AR-mediated effects are not the whole story when it comes to anabolic steroid activity in the body. There are still a host of other effects that have little to nothing at all to do with AR, known as non-AR dependent effects, which include central nervous system stimulation and a host of other anabolic and

potentially anabolic activities. But that still leaves us with the question of why our gains seem to slow down after a few cycles, and why we need to keep upping the dose. In truth, the answer probably has more to do with the body attempting to return to homeostasis through other mechanisms than it has with the androgen receptor per se. Still, if you’re worried about your androgen receptors you can try...no promises..some L-Carnitine L-Tartrate, a nutritional supplement that has been shown to increase androgen receptors (it was included in beastdrol, from NTBM for this exact reason).


Myth: Winstrol is the same whether you drink it or inject it.

Truth: When you pass a steroid through your liver it’s subject to a different metabolism to that of steroids injected directly into the muscle. In the case of Winstrol (Stanozolol), this includes greater interaction with SHBG, which lowers the amount of that carrier protein (allowing you to free up more androgen in the body). However, overall you get greater protein synthesis with the injectable route of administration, but both methods have their advantages. https://thinksteroids.com/articles/winstrol-oral-vs-injectable/ You can read more here, but injecting winstrol is called a first liver bypass. Seems science says injection is better. I have not found this to be so.


Myth: If you inject double the amount of testosterone you have twice as much in your bloodstream.

Truth: Again, this one seems to make a lot of sense. If you inject 600 milligrams of testosterone you should have twice as much in your blood as you would injecting 300 milligrams. But this isn’t how it works. The dose you’re taking isn’t equivalent to the blood plasma levels you’ll achieve – nor will doubling the dose necessarily double your blood plasma levels of testosterone. When scientists compared a 300 milligram shot of testosterone to a 600 milligram shot, they found that the 300mg weekly shot will get a normal male to approximately 1,345 ng/dl, while a 600mg weekly shot will get a normal male to 2,370 ng/dl (less than double the amount achieved with half the dose).


Myth: Testosterone is testosterone.

Truth: Although this seems like something that can’t be a myth, it is. Methyltestosterone (oral testosterone) converts to a much more potent version of oestrogen (methylestrogen) in the liver, while injectable testosterone does not. And, even the injectable testosterones have their differences; short esters convert to less oestrogen than longer esters. And, just to make things even more complicated, the patches and gels are convert to dihydrotestosterone to a much greater degree than either the injectable or oral testosterones.
Both oestrogen( I like to use the O...it is old school Oxford U!) and dihydrotestosterone have profound physiological effects on the body and, because every type of testosterone doesn’t convert to them equally, we find varying effects depending on the one we’re using. Oestrogen promotes greater adipose (fat) gain, but also greater muscle gain, while DHT gives a harder, more quality look to the physique. Still, although we see this “testosterone is testosterone” myth repeated (online
mostly) we also see top-flight bodybuilders taking advantage of the differences in various testosterones – long esters for bulking, short esters for cutting. So it’s not only the route of administration (oral, transdermal or injectable) that matters, but also the ester of the injectable versions. Testosterone is, strangely, not just testosterone.


Myth: Steroids don’t work unless you train and eat properly.

Truth: We’d love this to be true, wouldn’t we? But the truth is that numerous studies conducted on burn victims, the elderly and other special populations, tell us that anabolic steroids will work to build muscle even in the most catabolic conditions, and in the absence of training. We see the same thing with people who have terrible training programmes but continue to make gains because they’re juicing. For optimal results, steroids are added to a proper programme and diet, but they’ll help even if you’re doing everything wrong. Hey, I didn’t make the rules, I’m just telling you what the science says! Burn victims have sat in bed eating nasty ass hospital food and packed on some muscle. Sorry again ok?

Myth: The injection site doesn’t matter.

Truth: A study conducted by Minto et. al. in 1997 examined the differences between injecting in the deltoid (shoulder) versus the gluteus (butt). While we’d logically assume that if we’re injecting 100mgs of Nandrolone into the body it shouldn’t matter where the injection site is, but in reality nothing could be further from the truth. Injecting in the gluteus results in significantly higher blood plasma levels of the anabolic steroid in question. Put more simply, the men who received gluteal injections in the Minto study ended up with more Nandrolone in their bloodstream! In practical terms, this means if we’re going to inject one ml of anabolic steroid per week, we’d probably be getting the best results by alternating butt cheeks (or quads). For the guys doing a few ml per day, they’re going to have to stick themselves anywhere they’ve still got a piece of unscarred skin showing! Notice I said DECA...no ..sorry I said nandrolone but I meant deca. Well we always talk about those deep IM injections to leave the depot, to be slowly released. For some reason these long chain esters do better in bigger muscles. So lets do this. Long chains in the ass. TestE and cyp..where ever you want. Sus has some long chains and short chains...so if you really want to feel the shorts go delts...want to feel the decoanate go glutes




Myth: As long as the milligram amount is the same, the concentration doesn’t matter.

Truth: It seems like a reasonable assumption to say that a 100mg shot of Nandrolone is going to hit you the same, regardless of whether it’s 4ml of 25mg or 1ml of 100mg, right? Wrong. Minto (the guy from the myth above) took a look at Nandrolone volume per injection and compared it to final blood plasma levels, and found that the more highly concentrated shot (100mg in 1ml of oil versus 4ml) actually produced a higher concentration of steroid in the bloodstream. Why because the the less oil per mg means quicker crystal deposit, but this could also mean more pip. So for deca stay at 200mg/ml gear.


Myth: Equipoise (Bolednone) works similarly to Deca-Durabolin (Nandrolone).

Truth: These two anabolic steroids have been considered interchangeable in cycles since the first series of Underground Steroid Handbook updates were published (they were a newsletter, written by the late Dan Duchaine). When Dan first introduced Equipoise to the bodybuilding world, he stated that a quick look at it’s structure revealed that it probably worked something like Deca. Well, nothing could be further from the truth.
Deca is a progestin, an anabolic steroid derived from 19-nor testosterone, while Equipoise is simply testosterone with an additional double bond. If we look at their characteristics (conversion to oestrogenic metabolites, conversion to 5a-reduced metabolites), we find very few similarities between the two drugs. And while Equipoise has a reputation for increasing appetite, Deca lays claim to healing injuries and relieving joint pain. Deca is also likely to cause a bit more water retention at high mgs than Equipoise, although, when we talk in those kinds of terms we really start getting into a very subjective realm. The latest deca info is loking into the fact that deca causes increases of synovial fluid.
In reality deca and eq are pretty dissimilar, but because of a single error, made a couple of decades ago, we still see them being used interchangeably in cycles.

Ok my boys...you have a lot of reading to do, and I am heading back to costa rica. Hopefull Jake can answer questions for newbies! Take care all!
DrB

Lucky you, have a Great Trip!
 

stretch512

Member
Very useful and relevant information that we want and are interested in. Just wondering drb your thoughts on any research data on LVH (Left Ventricle Hypertrophy) caused from long term use of AAS? I'm wondering if there may be a differentiating from long term therapeutical dosing versus long term supraphysiological dosing. I'm hoping that there may be something out there that shows low dosing, even for prolonged periods do not have impact on the shaping of the left ventricle, but of course that's me wishing for that data so that I can tell myself "We're good to go with low dosing". Just curios if you've seen more conclusive data on this.

Very good question - I'm also interested to hear what the consensus is on this.
 

Jake

Active member
Very useful and relevant information that we want and are interested in. Just wondering drb your thoughts on any research data on LVH (Left Ventricle Hypertrophy) caused from long term use of AAS? I'm wondering if there may be a differentiating from long term therapeutical dosing versus long term supraphysiological dosing. I'm hoping that there may be something out there that shows low dosing, even for prolonged periods do not have impact on the shaping of the left ventricle, but of course that's me wishing for that data so that I can tell myself "We're good to go with low dosing". Just curios if you've seen more conclusive data on this.

DrB is on a plane.We won't hear from him for a while. I think that this condition is mostly a pre disposed genetic situation. It usually happens to the fast twitch guys, or the ectos. I would not go so far as to say aas has anything to with it, but there may be a causal relationship simply due to the fact that guys on aas are usually pushing their hearts harder. Most 100,200 yard sprinters will get this no matter if they juiced or not. It is almost like you lifting heavy weight. You keep pushing and pushing heavy and heavier so the muscle responds. Same goes for the heart. It is a muscle and in particular the left ventricle is the chamber where the blood is already oxygenated and now has to get out to the body by the strongest muscle possible...which is te left ventricle.
Plus it can simply be inherited.
 
DrB is on a plane.We won't hear from him for a while. I think that this condition is mostly a pre disposed genetic situation. It usually happens to the fast twitch guys, or the ectos. I would not go so far as to say aas has anything to with it, but there may be a causal relationship simply due to the fact that guys on aas are usually pushing their hearts harder. Most 100,200 yard sprinters will get this no matter if they juiced or not. It is almost like you lifting heavy weight. You keep pushing and pushing heavy and heavier so the muscle responds. Same goes for the heart. It is a muscle and in particular the left ventricle is the chamber where the blood is already oxygenated and now has to get out to the body by the strongest muscle possible...which is te left ventricle.
Plus it can simply be inherited.

Thanks Jake.......yeah that's kind of what I thought. We still haven't seen anything really conclusive if AAS has been a real contributor to the condition or not, but I wanted to see if you or your dad had seen anything newer and more conclusive. Thanks for your response. I'm one of those fast twitch guys, so I need to keep aware of this.
 

RickRock

Community Leader
VIP Moderator
Very very good information. There's a,lot of bro science out there that's been passed around as truth which is not the truth at all. I agree with everything Said Drb!
 

SpikedEggnog

Active member
There are few drugs in the world that are subjected to as much misinformation as anabolic steroids. When athletes were first using anabolic steroids in the 1930s > ’50s to break world records, the scientific and medical community simultaneously published studies that told us that they didn’t work. During that time the only legitimate information about these drugs was coming from the front lines; the weightlifters (and later bodybuilders) who had tried them and knew what they did. For decades there were scientists claiming anabolic steroids did not do anything to build muscle or strength and, to this day, the Physician’s Desk Reference says that they do not work to improve athletic performance.
Ultimately, medical science started playing catch up, but the damage was done and we steroid-users mostly turned a blind-eye to anything falling from the Ivory Towers of academia. And then came the Internet – where a hodge-podge of science and experience has been brought together to create some of the biggest steroid-myths we’ve ever seen. Unfortunately, since they come from other steroid users and not doctors, we’ve come to accept many of them as fact. And because the internet is the primary source of information on anabolic steroids at this point, the person who screams the loudest is the one who is most often repeated – right or wrong.

Myth: Steroids down-regulate your androgen receptors (this is why your first cycle is always your best).

Truth: The logic for this myth actually makes a lot of sense – receptor down-regulation is pretty obvious when you drink a cup of coffee every day for a month, then find you need to keep increasing the size to get the same “kick”. We see (and feel) this receptor downgrade with caffeine, clenbuterol, ephedrine and a ton of other stuff, so it’s logical to think that we’re seeing the same thing with steroids. Sadly, the science tells us otherwise. Steroids actually do the opposite – they up-regulate your androgen receptors. It’s wrong to think about androgen receptors as permanent receptacles for the androgen ligand (sort of like a fixed electrical outlet in your house). In reality, your androgen receptors are constantly being turned over. When unattached to an androgen they have a half life of approximately three hours and are ultimately replaced with new ones. However, in the presence of an androgen (i.e. when they’re attached), they become more sensitive, their half life is doubled and the amount of new receptors being formed also increases substantially. It’s also important to remember that AR-mediated effects are not the whole story when it comes to anabolic steroid activity in the body. There are still a host of other effects that have little to nothing at all to do with AR, known as non-AR dependent effects, which include central nervous system stimulation and a host of other anabolic and

potentially anabolic activities. But that still leaves us with the question of why our gains seem to slow down after a few cycles, and why we need to keep upping the dose. In truth, the answer probably has more to do with the body attempting to return to homeostasis through other mechanisms than it has with the androgen receptor per se. Still, if you’re worried about your androgen receptors you can try...no promises..some L-Carnitine L-Tartrate, a nutritional supplement that has been shown to increase androgen receptors (it was included in beastdrol, from NTBM for this exact reason).


Myth: Winstrol is the same whether you drink it or inject it.

Truth: When you pass a steroid through your liver it’s subject to a different metabolism to that of steroids injected directly into the muscle. In the case of Winstrol (Stanozolol), this includes greater interaction with SHBG, which lowers the amount of that carrier protein (allowing you to free up more androgen in the body). However, overall you get greater protein synthesis with the injectable route of administration, but both methods have their advantages. https://thinksteroids.com/articles/winstrol-oral-vs-injectable/ You can read more here, but injecting winstrol is called a first liver bypass. Seems science says injection is better. I have not found this to be so.


Myth: If you inject double the amount of testosterone you have twice as much in your bloodstream.

Truth: Again, this one seems to make a lot of sense. If you inject 600 milligrams of testosterone you should have twice as much in your blood as you would injecting 300 milligrams. But this isn’t how it works. The dose you’re taking isn’t equivalent to the blood plasma levels you’ll achieve – nor will doubling the dose necessarily double your blood plasma levels of testosterone. When scientists compared a 300 milligram shot of testosterone to a 600 milligram shot, they found that the 300mg weekly shot will get a normal male to approximately 1,345 ng/dl, while a 600mg weekly shot will get a normal male to 2,370 ng/dl (less than double the amount achieved with half the dose).


Myth: Testosterone is testosterone.

Truth: Although this seems like something that can’t be a myth, it is. Methyltestosterone (oral testosterone) converts to a much more potent version of oestrogen (methylestrogen) in the liver, while injectable testosterone does not. And, even the injectable testosterones have their differences; short esters convert to less oestrogen than longer esters. And, just to make things even more complicated, the patches and gels are convert to dihydrotestosterone to a much greater degree than either the injectable or oral testosterones.
Both oestrogen( I like to use the O...it is old school Oxford U!) and dihydrotestosterone have profound physiological effects on the body and, because every type of testosterone doesn’t convert to them equally, we find varying effects depending on the one we’re using. Oestrogen promotes greater adipose (fat) gain, but also greater muscle gain, while DHT gives a harder, more quality look to the physique. Still, although we see this “testosterone is testosterone” myth repeated (online
mostly) we also see top-flight bodybuilders taking advantage of the differences in various testosterones – long esters for bulking, short esters for cutting. So it’s not only the route of administration (oral, transdermal or injectable) that matters, but also the ester of the injectable versions. Testosterone is, strangely, not just testosterone.


Myth: Steroids don’t work unless you train and eat properly.

Truth: We’d love this to be true, wouldn’t we? But the truth is that numerous studies conducted on burn victims, the elderly and other special populations, tell us that anabolic steroids will work to build muscle even in the most catabolic conditions, and in the absence of training. We see the same thing with people who have terrible training programmes but continue to make gains because they’re juicing. For optimal results, steroids are added to a proper programme and diet, but they’ll help even if you’re doing everything wrong. Hey, I didn’t make the rules, I’m just telling you what the science says! Burn victims have sat in bed eating nasty ass hospital food and packed on some muscle. Sorry again ok?

Myth: The injection site doesn’t matter.

Truth: A study conducted by Minto et. al. in 1997 examined the differences between injecting in the deltoid (shoulder) versus the gluteus (butt). While we’d logically assume that if we’re injecting 100mgs of Nandrolone into the body it shouldn’t matter where the injection site is, but in reality nothing could be further from the truth. Injecting in the gluteus results in significantly higher blood plasma levels of the anabolic steroid in question. Put more simply, the men who received gluteal injections in the Minto study ended up with more Nandrolone in their bloodstream! In practical terms, this means if we’re going to inject one ml of anabolic steroid per week, we’d probably be getting the best results by alternating butt cheeks (or quads). For the guys doing a few ml per day, they’re going to have to stick themselves anywhere they’ve still got a piece of unscarred skin showing! Notice I said DECA...no ..sorry I said nandrolone but I meant deca. Well we always talk about those deep IM injections to leave the depot, to be slowly released. For some reason these long chain esters do better in bigger muscles. So lets do this. Long chains in the ass. TestE and cyp..where ever you want. Sus has some long chains and short chains...so if you really want to feel the shorts go delts...want to feel the decoanate go glutes




Myth: As long as the milligram amount is the same, the concentration doesn’t matter.

Truth: It seems like a reasonable assumption to say that a 100mg shot of Nandrolone is going to hit you the same, regardless of whether it’s 4ml of 25mg or 1ml of 100mg, right? Wrong. Minto (the guy from the myth above) took a look at Nandrolone volume per injection and compared it to final blood plasma levels, and found that the more highly concentrated shot (100mg in 1ml of oil versus 4ml) actually produced a higher concentration of steroid in the bloodstream. Why because the the less oil per mg means quicker crystal deposit, but this could also mean more pip. So for deca stay at 200mg/ml gear.


Myth: Equipoise (Bolednone) works similarly to Deca-Durabolin (Nandrolone).

Truth: These two anabolic steroids have been considered interchangeable in cycles since the first series of Underground Steroid Handbook updates were published (they were a newsletter, written by the late Dan Duchaine). When Dan first introduced Equipoise to the bodybuilding world, he stated that a quick look at it’s structure revealed that it probably worked something like Deca. Well, nothing could be further from the truth.
Deca is a progestin, an anabolic steroid derived from 19-nor testosterone, while Equipoise is simply testosterone with an additional double bond. If we look at their characteristics (conversion to oestrogenic metabolites, conversion to 5a-reduced metabolites), we find very few similarities between the two drugs. And while Equipoise has a reputation for increasing appetite, Deca lays claim to healing injuries and relieving joint pain. Deca is also likely to cause a bit more water retention at high mgs than Equipoise, although, when we talk in those kinds of terms we really start getting into a very subjective realm. The latest deca info is loking into the fact that deca causes increases of synovial fluid.
In reality deca and eq are pretty dissimilar, but because of a single error, made a couple of decades ago, we still see them being used interchangeably in cycles.

Ok my boys...you have a lot of reading to do, and I am heading back to costa rica. Hopefull Jake can answer questions for newbies! Take care all!
DrB

awesome, thanks drb
 
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